Difference between revisions of "Team:TP-CC San Diego"

Line 86: Line 86:
  
 
<body>
 
<body>
 
 
 
 
  
 
<div id="outer" data-aos="fade-right">
 
<div id="outer" data-aos="fade-right">
Line 104: Line 100:
 
</div>
 
</div>
  
 +
<div id="outer" data-aos="fade-right">
 +
<div id="inner">
 +
<div id="finalinner">
 +
<h2 style="margin-bottom: 0px;">PROJECT OVERVIEW</h2>
 +
<div id="borderBot"></div><br><br>
 +
<p style="margin-bottom: 15px;">
 +
Cancer, a genetic disease resulting in uncontrollable cell growth, is mostly caused by somatic mutations acquired throughout an individual’s lifetime.  Recently, it was discovered that some oncogenes resided on extrachromosomal DNA (ecDNA). Because ecDNA causes oncogene copy number to increase exponentially, utilizing CRISPR to create breaks in ecDNA decreases cancer cells’ replication speed.
 +
</p>
 +
<a class="findout"; style = "text-align: center" href="https://2017.igem.org/Team:TP-CC_San_Diego/Description">FIND OUT MORE</a>
 +
</div>
 +
</div>
 +
</div>
  
 
<script>
 
<script>

Revision as of 05:52, 11 October 2017

Project | ecDNA Cancer


PROJECT OVERVIEW



Cancer, a genetic disease resulting in uncontrollable cell growth, is mostly caused by somatic mutations acquired throughout an individual’s lifetime. Recently, it was discovered that some oncogenes resided on extrachromosomal DNA (ecDNA). Because ecDNA causes oncogene copy number to increase exponentially, utilizing CRISPR to create breaks in ecDNA decreases cancer cells’ replication speed.

FIND OUT MORE

PROJECT OVERVIEW



Cancer, a genetic disease resulting in uncontrollable cell growth, is mostly caused by somatic mutations acquired throughout an individual’s lifetime. Recently, it was discovered that some oncogenes resided on extrachromosomal DNA (ecDNA). Because ecDNA causes oncogene copy number to increase exponentially, utilizing CRISPR to create breaks in ecDNA decreases cancer cells’ replication speed.

FIND OUT MORE