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<h2 id="tip3" style="cursor: pointer;">MODEL FOR SECURITY</h2> | <h2 id="tip3" style="cursor: pointer;">MODEL FOR SECURITY</h2> | ||
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<h4> 对于一个有治疗意义的系统而言,我们最为关心的就是这个系统的安全性,而广义的安全性包含两个部分:安全性(do no harm),可控性,在此基础上,我们还考虑系统的有效性,这当然也是非常重要的。</h4> | <h4> 对于一个有治疗意义的系统而言,我们最为关心的就是这个系统的安全性,而广义的安全性包含两个部分:安全性(do no harm),可控性,在此基础上,我们还考虑系统的有效性,这当然也是非常重要的。</h4> |
Revision as of 07:52, 27 October 2017
Model
当代科学是严格化的科学,更是定量化的科学,我们已经不能满足于获取事物的定性性质,而要求去尽可能精确地衡量事物发展过程中量的变化,从中我们能获知事物隐藏在内部的性质,可以预知事物发展的方向。尤其是在这个由信息革命所引领的时代,数学能更加便捷地与其他学科进行交叉,这也促成了现在的新兴学科:计算生物学与生物信息学。工程类学科往往走在数学理论应用的最前沿,它们是数学进步的推动力,也是数学进步的受益者,而合成生物学,正是数学与生物工程的前沿阵地。
从加入到igem队伍时,我便在想:建模对于我们的队伍,我们的项目还有igem以及合成生物学本身,究竟能做些什么?
数学以其严格性和精确性,为我们提供了一个认识疾病和生物进程的绝佳工具。我们认为,model所能做的,应该不仅仅是单纯的模拟与验证我们的系统(当然那也很重要),关键在于:利用model的优势,去解决我们开展项目过程中,所遇到的问题,将会遇到的问题,去为整个队伍服务。所以我们定义了model的两个目标“2S”:
Model to serve, Model to solve
What did our model achieve?
We achieved 3 main aims in our modelling work
We introduced a novel ensemble modelling approach to iGEM and made this approach accessible to other iGEM teams by sharing our code.
We improved our understanding of our system and used real experimental data to improve our model, using network mechanism analysis and parameter relationship analysis.
Reciprocal differentiation model | cytokine-mediated inflammation in RA | Exploration |
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A Mathematical Model for the Reciprocal Differentiation of T Helper 17 Cells and Induced Regulatory T Cells
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a two-variable model for the interactions between pro-inflammatory and anti-inflammatory cytokines in rheumatoid arthritis READ MORE |
a mathematical representation of our SynNotch CAR-Tregs system and exploration of the previous two models
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